Google searches could unveil patterns in Black mortality rates across the US, according to a new study. Researchers found that those areas with greater levels of racism, as indexed by the proportion of Google searches containing the “n-word,” had higher mortality rates among Blacks. The study is the first to examine an Internet query-based measure of racism in relation to mortality risk.... Read more »
Chae, D., Clouston, S., Hatzenbuehler, M., Kramer, M., Cooper, H., Wilson, S., Stephens-Davidowitz, S., Gold, R., & Link, B. (2015) Association between an Internet-Based Measure of Area Racism and Black Mortality. PLOS ONE, 10(4). DOI: 10.1371/journal.pone.0122963
A buff-tailed bumblebee (Bombus terrestris
Buff-tailed bumblebees (Bombus terrestris) that have been infected by parasites seek out flowers with nicotine in their nectar, according to a new study by researchers at the Royal Holloway University of London and Queen Mary University of London, UK.
Apparently, the nicotine in the flowers slows the progression of disease in infected bees but has ... Read more »
Baracchi, D., Brown, M., & Chittka, L. (2015) Weak and contradictory effects of self-medication with nectar nicotine by parasitized bumblebees. F1000Research. DOI: 10.12688/f1000research.6262.1
How will ocean acidification from anthropogenic CO2 emissions affect marine life? Recent work studying a similar time during the Permian extinction 200 million years ago gives a clue.... Read more »
Clarkson MO, Kasemann SA, Wood RA, Lenton TM, Daines SJ, Richoz S, Ohnemueller F, Meixner A, Poulton SW, & Tipper ET. (2015) Ocean acidification and the Permo-Triassic mass extinction. Science (New York, N.Y.), 348(6231), 229-32. PMID: 25859043
Attention and impulsive behaviors found in attention deficit hyperactivity disorder (ADHD) can contribute to accident risk in children and adults.A recent study of adult drivers in France provides evidence for increased accident risk in adults with ADHD.Researchers at the Bordeaux University Hospital interviewed a series of adult drivers seen in the emergency department following a road traffic crash.A total of 777 eligible subjects completed assessments of accident information, distraction exposure and presence of ADHD, depression or anxiety disorders.The key findings from this study including the following:67 of the subjects (8.6%) were assigned to the ADHD categoryPresence of a depressive or anxiety disorder did not appear to be linked to risk for responsibility of a crashPresence of a distraction contributed to accident riskPresence of ADHD increased accident risk approximately two fold (Odds ratio = 2.18)Presence of both ADHD and a distraction exposure increased accident risk four fold (Odds ratio=4.37) The authors found that in the ADHD case group, use of ADHD medication was extremely rare. They note that this may be due to the underdiagnosis of ADHD in adults in France and in many other European countries.In the discussion section the authors point out:"Improved screening and diagnosis of ADHD in adults would enable adoption of care strategies that focus on reducing attention impairment and improving cognitive performance. These strategies, which may also include road safety awareness messages, are likely to reduce the risk of road crashes in patients with ADHD."This study in an important addition to the literature of ADHD and accident risk. It supports continued impairment for ADHD in some adults.Readers with more interest in this study can access the free full-text manuscript by clicking on the PMID link in the citation below.Walkers at sunset photo is from the author's files.Follow the author on Twitter WRY999El Farouki K, Lagarde E, Orriols L, Bouvard MP, Contrand B, & Galéra C (2014). The increased risk of road crashes in attention deficit hyperactivity disorder (ADHD) adult drivers: driven by distraction? Results from a responsibility case-control study. PloS one, 9 (12) PMID: 25536069... Read more »
El Farouki K, Lagarde E, Orriols L, Bouvard MP, Contrand B, & Galéra C. (2014) The increased risk of road crashes in attention deficit hyperactivity disorder (ADHD) adult drivers: driven by distraction? Results from a responsibility case-control study. PloS one, 9(12). PMID: 25536069
This announcement came out while I was at a conference last week–but I wanted to pass it along. This appears to be a big change in the way Phytozome works. And there will be down-time before it rolls out, starting May 1. I like to post major announcements from mailing lists because I know everyone […]... Read more »
Goodstein D. M., R. Howson, R. Neupane, R. D. Hayes, J. Fazo, T. Mitros, W. Dirks, U. Hellsten, N. Putnam, & D. S. Rokhsar. (2011) Phytozome: a comparative platform for green plant genomics. Nucleic Acids Research, 40(D1). DOI: http://dx.doi.org/10.1093/nar/gkr944
Under conditions of cell stress such as nutrient deprivation or as a result of the accumulation of damaged organelles and misfolded proteins a lysosomal pathway is induced which degrades proteins as well organelles independent of the proteasomal pathway. The core machinery of this pathway -termed autophagy from the Greek auto-, "self" and phagein, "to eat” (literally “selfeating”) - was discovered in genetic screens of yeast for genes required for survival under nutrient starvation and since been shown to be conserved in mammalian and plant cells alike, providing metabolic substrates in the absence of external sources as well as contributing to cell survival by degrading non-functional organelles.In the context of viral infection therefore the induction of autophagy can have both pro- as well as antiviral effects. Indeed, a number of viral proteins such as the coronaviral nsp-6 or PLP2 proteins both induce the formation of autophagosomes or autophagosome-like structures whilst preventing the fusion of these structures with the lysosome whilst the genome of other viruses encode for protein(s) which might prevent the fusion of autophagosomes whilst not inducing the formation of autophagosomes.Excessive autophagy in contrast, induces lipid depletion of the ER and/or nuclear membrane, and inducing a process termed autosis characterized by ER fragmentation, which may be distinct from “classical” autophagy induced cell death, the latter being induced by the release of Cathepsin-D and subsequent activation of caspases.In contrast to autosis, which may or may not involve the induction of ER stress response, the release of Cathepsin-D increases the release of Cytochrome C and subsequent activates Caspase-3/-7 thus inducing the depolarization of the mitochondrial membrane, linking defective autophagy to Caspase-dependent apoptosis.
Here the induction of autophagy is discussed as a result from the expression of the EBV LMP-1 and LMP-2A proteins. In the context of viral infection both LMP-1 and LMP-2A co-localize to lipid rafts, indicating that lipid rafts are the membrane source for the autophagic vesicles induced by both proteins, similar to HTLV-1 Tax. Since the expression of LMP-1 induces the ER stress response, it might be possible that LMP-2A increases the clearance of LMP-1 by enhancing LMP-1 induced autophagy. It remains to be seen if the combined expression of both LMP-1 and LMP-2A in the absence of other viral proteins such as EBNA-1 induces autophagic cell death or autosis due to lipid depletion. ... Read more »
Xu T, Nicolson S, Denton D, & Kumar S. (2015) Distinct requirements of Autophagy-related genes in programmed cell death. Cell death and differentiation. PMID: 25882046
Geng Y, Kohli L, Klocke BJ, & Roth KA. (2010) Chloroquine-induced autophagic vacuole accumulation and cell death in glioma cells is p53 independent. Neuro-oncology, 12(5), 473-81. PMID: 20406898
Pucer A, Castino R, Mirković B, Falnoga I, Slejkovec Z, Isidoro C, & Lah TT. (2010) Differential role of cathepsins B and L in autophagy-associated cell death induced by arsenic trioxide in U87 human glioblastoma cells. Biological chemistry, 391(5), 519-31. PMID: 20302512
Xi X, Zhang X, Wang B, Wang T, Wang J, Huang H, Wang J, Jin Q, & Zhao Z. (2013) The interplays between autophagy and apoptosis induced by enterovirus 71. PloS one, 8(2). PMID: 23437282
Bhoopathi P, Chetty C, Gujrati M, Dinh DH, Rao JS, & Lakka S. (2010) Cathepsin B facilitates autophagy-mediated apoptosis in SPARC overexpressed primitive neuroectodermal tumor cells. Cell death and differentiation, 17(10), 1529-39. PMID: 20339379
Hah YS, Noh HS, Ha JH, Ahn JS, Hahm JR, Cho HY, & Kim DR. (2012) Cathepsin D inhibits oxidative stress-induced cell death via activation of autophagy in cancer cells. Cancer letters, 323(2), 208-14. PMID: 22542809
Liu Y, & Levine B. (2015) Autosis and autophagic cell death: the dark side of autophagy. Cell death and differentiation, 22(3), 367-76. PMID: 25257169
Muñoz-Pinedo, C., & Martin, S. (2014) Autosis: a new addition to the cell death tower of babel. Cell Death and Disease, 5(7). DOI: 10.1038/cddis.2014.246
Liu Y, Shoji-Kawata S, Sumpter RM Jr, Wei Y, Ginet V, Zhang L, Posner B, Tran KA, Green DR, Xavier RJ.... (2013) Autosis is a Na ,K -ATPase-regulated form of cell death triggered by autophagy-inducing peptides, starvation, and hypoxia-ischemia. Proceedings of the National Academy of Sciences of the United States of America, 110(51), 20364-71. PMID: 24277826
Busson P, McCoy R, Sadler R, Gilligan K, Tursz T, & Raab-Traub N. (1992) Consistent transcription of the Epstein-Barr virus LMP2 gene in nasopharyngeal carcinoma. Journal of virology, 66(5), 3257-62. PMID: 1313931
Fotheringham JA, & Raab-Traub N. (2015) Epstein Barr-Virus Latent Membrane Protein 2 Induces Autophagy to Prevent Cell Death. Journal of virology. PMID: 25878108
Longnecker R, & Kieff E. (1990) A second Epstein-Barr virus membrane protein (LMP2) is expressed in latent infection and colocalizes with LMP1. Journal of virology, 64(5), 2319-26. PMID: 2157888
Granato M, Santarelli R, Farina A, Gonnella R, Lotti LV, Faggioni A, & Cirone M. (2014) Epstein-barr virus blocks the autophagic flux and appropriates the autophagic machinery to enhance viral replication. Journal of virology, 88(21), 12715-26. PMID: 25142602
Li Y, Zhang L, Zhou J, Luo S, Huang R, Zhao C, & Diao A. (2015) Nedd4 E3 ubiquitin ligase promotes cell proliferation and autophagy. Cell proliferation. PMID: 25809873
Kuang E, Qi J, & Ronai Z. (2013) Emerging roles of E3 ubiquitin ligases in autophagy. Trends in biochemical sciences, 38(9), 453-60. PMID: 23870665
Abrahamsen H, Stenmark H, & Platta HW. (2012) Ubiquitination and phosphorylation of Beclin 1 and its binding partners: Tuning class III phosphatidylinositol 3-kinase activity and tumor suppression. FEBS letters, 586(11), 1584-91. PMID: 22673570
Xia P, Wang S, Du Y, Zhao Z, Shi L, Sun L, Huang G, Ye B, Li C, Dai Z.... (2013) WASH inhibits autophagy through suppression of Beclin 1 ubiquitination. The EMBO journal, 32(20), 2685-96. PMID: 23974797
Naon D, & Scorrano L. (2014) At the right distance: ER-mitochondria juxtaposition in cell life and death. Biochimica et biophysica acta, 1843(10), 2184-94. PMID: 24875902
Frappier L. (2012) Contributions of Epstein-Barr nuclear antigen 1 (EBNA1) to cell immortalization and survival. Viruses, 4(9), 1537-47. PMID: 23170171
I recently came across the paper by Martina Barnevik Olsson and colleagues  (open-access) and their rather interesting take on the issue of optimal outcome and autism (see here for some background on this concept).Based on the idea that a diagnosis of autism or autism spectrum disorder (ASD) might not be as immutable as perhaps once thought (as in 'no longer meeting the diagnostic criteria for the condition'), Barnevik Olsson et al reported that loss of the autism/ASD label does not necessarily translate into typical developmental service being resumed. Indeed, that the concept of ESSENCE (Early Symptomatic Syndromes Eliciting Neurodevelopmental Clinical Examinations) or autism+  if you prefer, might still influence clinical presentation and the subsequent continued "need of support, educationally, from a neurodevelopmental and a medical point of view."Following 17 children originally diagnosed with ASD who "recovered from autism" after behavioural intervention, researchers took various 'readings' at follow-up points covering "the child’s daily functioning, school situation, and need of support." Alongside a parental interview, authors also used the Vineland Adaptive Behavior Scales (VABS) and the "Autism – Tics, Attention-deficit/hyperactivity disorder (AD/HD), and other Comorbidities (A-TAC) telephone interview" to see whether loss of the ASD label meant 'symptom-free'.Results, and a long quote coming up:"At the new follow-up around age 10 years, all the children had major behavioral and/or academic problems. Of the 13 children with social interaction problems in the semistructured parental interview, 12 also had repeated tantrums, nine had difficulties with hyperactivity or impulsivity, and two with passivity. Eleven of the children had difficulties concentrating, and ten had speech problems. Hence, it was evident that a majority of the children had problems in several different domains." The authors note that many of these presented symptoms fall under the umbrella term of ESSENCE. Further, that of the 14 children who's parents were available for the A-TAC interview, three of them were again considered to meet the criteria for ASD and "another six had pronounced subthreshold ASD symptoms."Hopefully without the 'I told you so' attitude coming out to much, I have covered the issue of optimal outcome not necessarily translating into 'symptom-free' before on this blog (see here). Once again, I'm not trying to reverse my excitement about the original results from Deborah Fein and colleagues  but rather pushing the idea that autism is very often much more than the sum of the triad/dyad of characteristics which we use (see here). Further research from Fein and colleagues  has also hinted that functioning outside of the label of autism does not necessarily translate into a complete loss of certain issues too as has data from other groups.Fluidity in the presentation of autistic traits is still something of real interest to autism research including that reaching into adulthood (see here). The Barnevik Olsson results add to that interest, incorporating the idea that quite a bit of the heightened comorbidity potentially present alongside a diagnosis of ASD may very well have some pretty significant effects on a person in terms of daily functioning and onwards issues affecting quality of life . In policy terms, what this means is that just because a child (or adult) drops off the autism spectrum symptom threshold wise, may not necessarily translate into no additional help and support being required. Indeed, as per the increasing interest in the DSM-5 criteria change for autism (see here) and the rise and rise of labels such as social communication disorder (SCD) one wonders how many optimal outcomers will merely fall out of the autism/ASD label and into the SCD category?Music: Seasick Steve - Summertime Boy.---------- Barnevik Olsson M. et al. “Recovery” from the diagnosis of autism – and then? Neuropsychiatric Disease and Treatment. 2015. 11: 999-1005. Fein D. et al. Optimal outcome in individuals with a history of autism. J Child Psychol Psychiatry. 2013 Feb;54(2):195-205. Orinstein AJ. et al. Social Function and Communication in Optimal Outcome Children and Adolescents with an Autism History on Structured Test Measures. J Autism Dev Disord. 2015 Mar 11. Gotham K. et al. Depressive and anxiety symptom trajectories from school age through young adulthood in samples with autism spectrum disorder and developmental delay. J Am Acad Child Adolesc Psychiatry. 2015 May;54(5):369-376.e3.----------Barnevik Olsson, M., Westerlund, J., Lundström, S., Giacobini, M., Fernell, E., & Gillberg, C. (2015). “Recovery” from the diagnosis of autism – and then? Neuropsychiatric Disease and Treatment DOI: 10.2147/NDT.S78707... Read more »
Barnevik Olsson, M., Westerlund, J., Lundström, S., Giacobini, M., Fernell, E., & Gillberg, C. (2015) “Recovery” from the diagnosis of autism – and then?. Neuropsychiatric Disease and Treatment, 999. DOI: 10.2147/NDT.S78707
A patient with a moderate or severe traumatic brain injury is likely to have structural brain changes that are associated with an older brain than his/her actual age.... Read more »
Cole JH, Leech R, Sharp DJ, & Alzheimer's Disease Neuroimaging Initiative. (2015) Prediction of brain age suggests accelerated atrophy after traumatic brain injury. Annals of Neurology, 77(4), 571-81. PMID: 25623048
The U.S. Food and Drug Administration recently admonished TauMark™, a brain diagnostics company, for advertising brain scans that can diagnose chronic traumatic encephalopathy (CTE), Alzheimer's disease, and other types of dementia. The Los Angeles Times reported that the FDA ordered UCLA researcher Dr. Gary Small and his colleague/business partner Dr. Jorge Barrio to remove misleading information from their company website (example shown below).CTE has been in the news because the neurodegenerative condition has been linked to a rash of suicides in retired NFL players, based on post-mortem observations. And the TauMark™ group made headlines two years ago with a preliminary study claiming that CTE pathology is detectable in living players (Small et al., 2013).The FDA letter stated:The website suggests in a promotional context that FDDNP, an investigational new drug, is safe and effective for the purpose for which it is being investigated or otherwise promotes the drug. As a result, FDDNP is misbranded under section 502(f)(1) of the FD&C Act... [18F]-FDDNP1 is a molecular imaging probe that crosses the blood brain barrier and binds to several kinds of abnormal proteins in the brain. When tagged with a radioactive tracer, FDDNP can be visualized using PET (positron emission tomography).Despite what the name of the company implies, FDDNP is not an exclusive tau marker. FDDNP may bind to tau protein [although this is disputed],2 but it also binds to beta-amyloid, found in the clumpy plaques that form in the brains of those with Alzheimer's disease. Tau is found in neurofibrillary tangles, also characteristic of Alzheimer's pathology, and seen in other neurodegenerative tauopathies such as CTE.The big deal with this and other radiotracers is that the pathological proteins can now be visualized in living human beings. Previously, an Alzheimer's diagnosis could only be given at autopsy, when the post-mortem brain tissue was processed to reveal plaques and tangles. So PET imaging is a BIG improvement. But still, a scan alone is not completely diagnostic, as noted by the Alzheimer's Association:Even though amyloid plaques in the brain are a characteristic feature of Alzheimer's disease, their presence cannot be used to diagnose the disease. Many people have amyloid plaques in the brain but have no symptoms of cognitive decline or Alzheimer's disease. Because amyloid plaques cannot be used to diagnose Alzheimer's disease, amyloid imaging is not recommended for routine use in patients suspected of having Alzheimer's disease.from TauMark's old websiteThere are currently three FDA-approved molecular tracers that bind to beta-amyloid: florbetapir, flutemetamol, and florbetaben (note that none of these is FDDNP). But the big selling point of TauMark™ is (of course) the tau marker part, which would also label tau in the brains of individuals with CTE and frontotemporal dementia, diseases not characterized by amyloid plaques. But how can you tell the difference, when FDDNP targets plaques and tangles (and prion proteins, for that matter)?A new study by the UCLA team demonstrated that the distribution of FDDNP labeling in the brains of Alzheimer's patients differs from that seen in a selected group of former NFL players with cognitive complaints (Barrio et al., 2015). These retired athletes (and others with a history of multiple concussions) are at risk of developing the brain pathology known as chronic traumatic encephalopathy. from Fig. 1 (Barrio et al., 2015). mTBI = mild traumatic brain injury, or concussion. T1 to T4 = progressive FDDNP PET signal patterns.It's a well-established fact that brains with Alzheimer's disease, frontotemporal lobar degeneration, or Lou Gehrig's disease (for example) all show different patterns of neurodegeneration, so why not extend this to CTE? This may seem like a reasonable approach, but there are problems with some of the assumptions.Perhaps the most deceptive claim is that “TauMark owns the exclusive license of the first and only brain measure of tau protein...” Au c... Read more »
Barrio, J., Small, G., Wong, K., Huang, S., Liu, J., Merrill, D., Giza, C., Fitzsimmons, R., Omalu, B., Bailes, J.... (2015) In vivo characterization of chronic traumatic encephalopathy using [F-18]FDDNP PET brain imaging. Proceedings of the National Academy of Sciences, 201409952. DOI: 10.1073/pnas.1409952112
"My wine is good to me, it helps me pass the time. And my good old buddy whiskey keeps me warmer than the sunshine," Aloe Blacc – I need a dollar, 2011.Psychologists have documented a striking increase in references to alcohol and heavy drinking in the lyrics of UK chart music. They warn this could mean that attempts to control the direct advertising of alcohol to young people will be in vain, as pop music is effectively spreading a positive message on the drinks companies' behalf.Katherine Hardcastle and her colleagues analysed all songs (611 in total) that reached a top 10 UK chart position in the years 1981, 1991, 2001 and 2011. The proportion of songs that referenced alcohol in their lyrics was 5.8, 2.1, 8.1 and 18.5 per cent, respectively across these years. The researchers also looked to see whether the references to alcohol and drinking carried negative, neutral or positive connotations. References were mixed in 1981; all positive in '91 (though this was the year with the lowest number of alcohol references); more negative and neutral than positive in 2001; while in 2011, the positive and neutral references (22 songs) far outnumbered the negative references (4).From Hardcastle et al. 2015Why are alcohol references on the increase in the British pop charts? Hardcastle and her co-authors think it has to do with the influence of US acts. Alcohol references are even more prevalent in the USA chart (23.7 per cent of songs in 2008) and songs by US acts in the UK chart contained more alcohol references than songs by British acts. References to booze and drinking were highest in Urban music (R&B, hip-hop and rap) – a genre largely originating in the US. "Today's urban music scene is dominated by US artists such as Jay-Z and Alicia Keys," the researcher said, "with many artists from the UK music scene attempting to emulate the sounds and styles of their American counterparts."This study cannot answer the question of whether mentions of alcohol (especially positive ones) in pop music encourages more alcohol abuse among young listeners. However, the researchers argue there is reason to think it might. They point to the influence of non-conscious priming (ideas can influence our behaviour without us realising it) and past research showing that people drink more when in a bar that's playing music with alcohol-related lyrics. Moreover, teenagers' beliefs about what's "normal" drinking behaviour will likely be influenced by what they hear from the singers they admire. "A greater understanding of the impacts of alcohol-related popular music is urgently needed," the researchers concluded._________________________________ Hardcastle, K., Hughes, K., Sharples, O., & Bellis, M. (2015). Trends in alcohol portrayal in popular music: A longitudinal analysis of the UK charts Psychology of Music, 43 (3), 321-332 DOI: 10.1177/0305735613500701 --further reading--Pop music is getting sadderPost written by Christian Jarrett (@psych_writer) for the BPS Research Digest.
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Hardcastle, K., Hughes, K., Sharples, O., & Bellis, M. (2013) Trends in alcohol portrayal in popular music: A longitudinal analysis of the UK charts. Psychology of Music, 43(3), 321-332. DOI: 10.1177/0305735613500701
A team of Belgian philosophers and plant biotechnologists have turned to cognitive science to explain why opposition to genetically modified organisms (GMOs) has become so widespread, despite positive contributions GM crops have made to sustainable agriculture. They argue that the human mind is highly susceptible to the negative and often emotional representations put out by certain environmental groups and other opponents of GMOs. The researchers urge the general public to form opinions on GMOs on a case-by-case basis, thereby not focusing on the technology but on the resulting product.... Read more »
Blancke, S., Van Breusegem, F., De Jaeger, G., Braeckman, J., & Van Montagu, M. (2015) Fatal attraction: the intuitive appeal of GMO opposition. Trends in Plant Science. DOI: 10.1016/j.tplants.2015.03.011
MedicalResearch.com Medical Research Interviews and News
MedicalResearch.com Interview with: Stefan Zeuzem, MD Professor of Medicine Chief Department of Medicine Goethe University Hospital Frankfurt Medical Research: What is the background for this study? What are the main findings? Dr. Zeuzem: Interferon- and ribavirin-free regimens are needed to … Continue reading →
The post Single Pill Combination Therapy For Some Hepatitis C Subtypes appeared first on MedicalResearch.com Medical Research Interviews and News.
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MedicalResearch.com Interview with: Stefan Zeuzem, MD. (2015) Single Pill Combination Therapy For Some Hepatitis C Subtypes. MedicalResearch.com. info:/
This preprint from Trudy Mackay‘s group at NC State uses a recombinant inbred series of flies known as the Drosophila Genome Reference Panel (DGRP) to evaluate how genetic variation affects transcription. To do this they extracted RNA from whole flies, pooled from males and females of each of the 192 recombinant inbred fly lines. They fit…... Read more »
Wen Huang, Mary Anna Carbone, Michael Magwire, Jason Peiffer, Richard Lyman, Eric Stone, Robert Anholt, & Trudy Mackay. (2015) Genetic Basis of Transcriptome Diversity in Drosophila melanogaster. bioRxiv. info:/10.1101/018325
A summary of our recent paper on the role of the TORC1 complex in drosophila muscle function... Read more »
Hatfield, I., Harvey, I., Yates, E., Redd, J., Reiter, L., & Bridges, D. (2015) The role of TORC1 in muscle development in Drosophila. Scientific Reports, 9676. DOI: 10.1038/srep09676
MedicalResearch.com Medical Research Interviews and News
MedicalResearch.com Interview with: Mary J Hamel, M.D. Chief, Strategic and Applied Sciences Unit, And Deputy Branch Chief for Science, CDC Malaria Branch US Centers for Disease Control and Prevention 1600 Clifton Rd, NE, MS A06 Atlanta GA 30333 Dr. Hamel … Continue reading →
The post Malaria Vaccine Has Potential To Contribute To Disease Control appeared first on MedicalResearch.com Medical Research Interviews and News.
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MedicalResearch.com Interview with: Mary J Hamel, M.D. (2015) Malaria Vaccine Has Potential To Contribute To Disease Control . MedicalResearch.com. info:/
MedicalResearch.com Medical Research Interviews and News
MedicalResearch.com Interview with Jay Giri, MD MPH Director, Peripheral Intervention Assistant Professor of Clinical Medicine University of Pennsylvania MedicalResearch: What is the background for this study? What are the main findings? Dr. Giri: Carotid artery stents are placed by vascular … Continue reading →
The post Carotid Artery Stenting: Comparison Of Embolic-Prevention Devices appeared first on MedicalResearch.com Medical Research Interviews and News.
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MedicalResearch.com Interview with Jay Giri, MD MPH. (2015) Carotid Artery Stenting: Comparison Of Embolic-Prevention Devices. MedicalResearch.com. info:/
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MedicalResearch.com Interview with: Ojan Assadian, M.D., DTMH Professor for Skin Integrity and Infection Prevention Institute for Skin Integrity and Infection Prevention School of Human & Health Sciences University of Huddersfield Queensgate, Huddersfield UK MedicalResearch: What is the background for this … Continue reading →
The post Antibacterial Gloves May Reduce Cross Contamination In ICU Setting appeared first on MedicalResearch.com Medical Research Interviews and News.
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MedicalResearch.com Interview with:Ojan Assadian, M.D., DTMH. (2015) Antibacterial Gloves May Reduce Cross Contamination In ICU Setting. MedicalResearch.com. info:/
MedicalResearch.com Medical Research Interviews and News
MedicalResearch.com Interview with: Neel S. Madhukar Graduate student in the lab of Olivier Elemento, PhD, Associate Professor Head, Laboratory of Cancer Systems Biology Department of Physiology and Biophysics Institute for Computational Biomedicine Weill Cornell Medical College Medical Research: What is … Continue reading →
The post Leveraging Big Data to Accelerate Drug Discovery appeared first on MedicalResearch.com Medical Research Interviews and News.
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MedicalResearch.com Interview with:Neel S. MadhukarGraduate student in the lab of, & Olivier Elemento, PhD, Associate Professor. (2015) Leveraging Big Data to Accelerate Drug Discovery . MedicalResearch.com. info:/
When Sandy Hook happened, it was so shocking that to this day, some don’t actually believe it happened. Shortly after, something frustrating happened, the shooter was labeled with aspergers. This helped drive the mental health and violence connection to the point that Time came out with an article dispelling that myth. Even now according to new longitudinal study of delinquent youth, most psychiatric disorders – including depression — do not predict future violent behavior. The only exception is substance abuse and dependence.... Read more »
Elkington, K., Teplin, L., Abram, K., Jakubowski, J., Dulcan, M., & Welty, L. (2015) Psychiatric Disorders and Violence: A Study of Delinquent Youth After Detention. Journal of the American Academy of Child , 54(4), 302-31200000. DOI: 10.1016/j.jaac.2015.01.002
There is some excitement in the net about some news of Harold White’s experiment at NASA. I have uncovered it by chance at a forum. This is a well-frequented site with people at NASA posting on it and regularly updating about the work that they are carrying out. You can also have noticed some activity […]... Read more »
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